JACKEDFORUMS

IGF-1 DES

Des(1-3) Insulin-like Growth Factor 1 · DES(1-3)IGF-1 · IGF-1 DES(1-3) · des-IGF-1 · IGF-1 DES 1,3

Truncated IGF-1 analogue with roughly 10x potency and a 20-30 minute window of action.

Typical dose

100 mcg

Once daily

Range

50–150

mcg

Half-life

24 min

see Dosing

Evidence

Animal only

Intramuscular · Subcutaneous

What it is

IGF-1 DES is native IGF-1 with the first three amino acids of the N-terminus (glycine-proline-glutamate) removed, leaving a 67-residue peptide. That small deletion destroys most of the molecule's affinity for IGF binding proteins, so a much larger fraction of an injected dose stays free and can hit the IGF-1 receptor immediately.

The practical trade-off is potency without duration. In rodent work DES is roughly ten times more potent than intact IGF-1 on a per-microgram basis, but it is cleared from circulation in well under an hour. Users exploit that by injecting into or beside the muscle group they have just trained, on the theory that it acts locally before it degrades.

There are no human trials of des(1-3)IGF-1 as a physique drug. Everything past the animal literature is bodybuilding practice, and grey-market IGF-1 analogues vary enormously in purity and correct folding between suppliers.

How it works

In the body, IGF binding proteins sequester the overwhelming majority of circulating IGF-1 and release it slowly. Removing the N-terminal tripeptide wrecks the IGFBP contact site, so DES circulates unbound and binds the IGF-1 receptor directly. Receptor activation drives PI3K/Akt/mTOR signalling, which raises muscle protein synthesis, increases glucose uptake and pushes satellite cells into proliferation.

Because nothing is holding it in circulation, DES is also cleared very fast. Its effect is a short, sharp pulse rather than the broader systemic exposure of IGF-1 LR3, which escapes the binding proteins and is dosed once daily as a background compound. That is exactly why the community uses DES around training instead.

Dosing

Typical single dose100 mcg
Reported range50–150 mcg
FrequencyOnce daily
Injections per week7
Half-life~20-30 minutes in circulation (animal and in vitro clearance data); effectively gone within the hour
TimingWithin 15-30 minutes of finishing training, injected into or adjacent to the muscle group just worked; eat carbohydrate shortly after.
Typical run length4-6 weeks
RoutesIntramuscular, Subcutaneous
Molecular weight7372 Da
SequenceTLCGAELVDALQFVCGDRGFYFNKPTGYGSSSRRAPQTGIVDECCFRSCDLRRLEMYCAPLKPAKSA

Many users run it on training days only, which is 4-6 doses a week rather than 7. Doses above ~150 mcg per injection mostly buy more hypoglycaemia rather than more growth.

Reconstitution calculator

Pre-loaded with IGF-1 DES’s vial size, water volume and typical dose. Change anything.

Typical: 100 mcg · range 50–150

05101520253010 units0.3 mL insulin · U-100 · half-unit marks

Draw to

10 u

0.1 mL · 100 mcg

Concentration

1 mg/mL

10 mcg per unit

Doses per vial

10

Vial lasts

10 days

Draw to 10 units on the barrel.

Once mixed
Refrigerated at 2-8C and used within 2-3 weeks; IGF-1 analogues lose activity faster in solution than short-chain peptides, so do not batch-mix a month ahead.

Full calculator →
Suggested water1 mL per 1 mg vial
Common vial sizes1 mg

1 mL of diluent into a 1 mg vial gives 1000 mcg/mL, so 100 mcg is 10 units on a U-100 insulin syringe. IGF-1 analogues are more stable in 0.6% acetic acid than in bacteriostatic water; if you use bac water, expect a shorter usable life. Aim the stream down the vial wall and swirl, never shake, as this peptide is disulfide-folded and shear will denature it.

Storage

Before mixing

Sealed and dry at -20C it holds for 18-24 months; a few weeks at 2-8C is fine for a vial in use.

After mixing

Refrigerated at 2-8C and used within 2-3 weeks; IGF-1 analogues lose activity faster in solution than short-chain peptides, so do not batch-mix a month ahead.

Reported benefits

  • Sharp, short-lived spike in IGF-1 receptor signalling at the injection site
  • Roughly tenfold more potent than intact IGF-1 in animal models because it evades IGFBPs
  • Drives satellite cell proliferation and muscle protein synthesis
  • Increases local glucose and amino acid uptake into recently trained tissue
  • Short duration means less systemic exposure than IGF-1 LR3
  • Unlike exogenous GH it does not act on the pituitary directly, but IGF-1 is itself the main negative-feedback signal on GH release, so systemic exposure still suppresses your own GH - the very short half-life just keeps that suppression brief

Side effects

  • Hypoglycaemia within 20-30 minutes of dosing: shakiness, sweating, hunger, confusion
  • Injection-site pain, swelling or a temporary lump
  • Joint aches and carpal-tunnel-type numbness at higher doses
  • Mild fluid retention
  • Theoretical acceleration of any existing precancerous or cancerous tissue, since IGF-1 signalling is mitogenic
  • Poorly folded or underdosed grey-market product producing no effect at all, or an immune reaction

Do not use if

  • Active cancer, a history of cancer, or strong family history of hormone-sensitive tumours
  • Proliferative diabetic retinopathy or other proliferative eye disease
  • Concurrent insulin use without glucose monitoring, as the hypoglycaemic effects stack
  • Uncontrolled hypoglycaemia or a history of severe hypoglycaemic episodes

Evidence level — Animal only

Preclinical animal data — no meaningful human trials.

Not approved by the FDA or EMA for any indication, sold only as a research chemical, and prohibited at all times under the WADA code.

Commonly run with

IGF-1 DES FAQ

Is IGF-1 DES actually site-specific?+

Only partly. Local concentration around the injection is higher than elsewhere for a short window, but the peptide diffuses and enters circulation, which is why people get systemic hypoglycaemia after a delt injection. There is no human evidence that site injection produces localised growth.

DES or LR3?+

They are used differently, though the usual explanation overstates the pharmacokinetic gap. DES is a 20-30 minute pulse injected peri-workout. LR3 is dosed once daily and treated as the all-day systemic option, but the 20-30 hour half-life quoted for it comes from supplier literature rather than measurement — no human PK exists, and in rats labelled LR3 actually clears faster than native IGF-1. The real difference is that LR3 escapes the binding proteins and distributes systemically, so it is dosed as a background compound; DES is the shorter, sharper local pulse. Running both at once mostly compounds the hypoglycaemia and the cost.

Do I have to reconstitute it with acetic acid?+

No, but it helps. IGF-1 analogues are considerably more stable in dilute (0.6%) acetic acid than in bacteriostatic water. Bac water works and is what most people use, but plan to finish the vial in two to three weeks rather than two months.

Why do I feel dizzy and starving after injecting?+

That is hypoglycaemia from IGF-1 receptor and cross-reactive insulin receptor activation. Have carbohydrate available before you inject, dose after training rather than fasted, and drop the dose if it happens at 50-100 mcg.

How long should a run be?+

Four to six weeks is the usual block. There is no receptor-desensitisation data to justify a specific limit, so the ceiling is practical: cost, injection-site tolerance, and not wanting to keep a mitogenic signal elevated indefinitely.

References

  1. 1.Insulin-like growth factor-I (IGF-I) and especially IGF-I variants are anabolic in dexamethasone-treated ratsBiochemical Journal (1992) PMID 1371669
  2. 2.Insulin-like growth factor-I and its N-terminal modified analogues induce marked gut growth in dexamethasone-treated ratsJournal of Endocrinology (1992) PMID 1613443
  3. 3.Insulin-like growth factor-I and more potent variants restore growth of diabetic rats without inducing all characteristic insulin effectsBiochemical Journal (1993) PMID 7683875

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