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Blood Results at 8 weeks in

Hey man! Sure thing, all the information I’ve gotten on EQ and it’s estrogen suppression comes from Derek at More Plates More Dates and Leo and Longevity. They both have deep dives on EQ. Interesting talks.
 
Since everything seems to be going well with this cycle I’m hesitant to make any modifications, but wonder if for my next one which I plan to run basically the same thing but adding Proviron if it may be good to bring EQ dose down to 500 and run it the same with test at 500 to have estrogen at a more normal range to get the most gains I can from it. This would be ran in the fall.
 
Thanks @Ldog! This is one of the many reasons I really like you guys, particularly your initial comment, let alone excellent communication and quick processing. I doubt many sponsors provide suggestions that lowering a planned cycle dose may be best and equally effective. Great to also have a sponsor that also is about harm reduction and getting the most out of a cycle at a lower dose, rather than focusing solely on profit. 👍
 
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Thanks bro…I’ve always(GC as well) been an advocate for low doses…get the most out of the lowest dose before ramping up. Many guys jump up too much and too fast…you can get so much out of low-moderate doses…plus this mindset will allow you to play in this game long term.
 
I recently just found this video and certainly explains the crashing of my E2 without any AI, if EQ metabolizes in a way that interferes with normal test to E2 aromatase. I’m beginning to feel like the EQ could be hindering my gains as my test levels are definitely up there. No added anxiety really, but am starting to notice some reduction in libido as of recently. Given EQ stays in your system quite long, it sounds like starting a PCT 2 weeks after last pin may be too early unless I start gradually reducing the EQ now but keeping my test the same. In the fall I’m now considering just going more basic and cut the EQ and run just test and Proviron.

It seems like the method of using EQ has changed over the past decade where I believe it was more common to run it like deca so half or at least lower than your test dose. Still making good gains, but have to wonder if I dropped the EQ if that may be better as my estrogen is quite low per my blood results 8 weeks in.

Thoughts on maybe switching the fall cycle to NPP with test and Proviron? I have mild thinning starting in hair (typical in front sides receding and a little on top in middle of my head) so trying to hold on to it if I can, but have no problem shaving it once it becomes really noticeable.
 
That’s not a bad idea. It was suggested to me when I using Deca to stop it a month earlier then Test and wait additional 2 weeks before PCT (4 weeks total). The only thing I did different was blasted HCG 2 weeks before nolva and clomid.
Test and NPP but w/Tbol is what my next one will be.
 
EQ 4-6wks depending upon the testosterone ester until starting pct.

I really enjoy test npp proviron

I have been seeing people running EQ at 800-1200mg I have never heard of running it like decca? Where did this idea come from?

Sounds like your e2 is high but you said you crashed your e2 without an AI? How?
 
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I think it was more bro science 10 or more years ago where test was often 500 with EQ at 400. Now it seems most run the EQ ~double the test.

As for why E2 crashed I was surprised too as I am not running anything that can think of that would affect normal test to E2 aromatase. It was theorized (haven’t found medical papers discussing it yet), but appears EQ acts differently with how it interacts with aromatase resulting in decreased aromatase of test to E2.
 
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Shock6050 said:
It was theorized (haven’t found medical papers discussing it yet), but appears E2 acts differently with how it interacts with aromatase resulting in suppression of E2.
Click to expand...
I don’t understand how this would happen with EQ and test cyp?
Im really curious about this I love this kinda stuff.
I know that its not good for you but Iove to know what actually caused this to happen because im baffled.

No other compounds? No dietary supplements?
 
Nope nothing as far as compounds. Dietary supplements are just a multivitamin and fish oil. If you look above @PeteGrimkowsky, the video that talks about this in more depth is at MPMD (very cheesy podcast channel name I know but he does do a good job explaining based on science how certain compounds work). I think the video is ‘Legendary Steroid for Horses’. LOL. I think he usually has links to literature to back it up so will dig through that.
 
What’s also really interesting is that dbol and EQ are almost identical in chemical structure but have much different effects due to mode of delivery.
 
Just like EQ DBOL and Primo all extremely similar just little tweaks to the compounds structure and you get 3 dramatically different compounds.
Even tbol is just dbol with the chlorol ring added I believe and it stops it from aromatase just with that addition.

Its amazing how close alot of compounds are and as I have been filling ugmuscle with AAS profiles I have seen the trend that all compounds come from just a few base compounds.

Testosterone, nandrolone or nortestosterone, methyltestosterone and dihydrotestosterone, dihydroboldenone these seem to be the dominant chemical structures that they start with and create other compounds from
 
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So from just a few quick searches it appears that the big picture is potentially this: EQ metabolites interact with aromatase and convert to estrone (or possibly a different type of estrogen that is not E2) and essentially blocks testosterone’s ability to convert to E2. Quite concerning given an adequate test/E2 ratio is needed for optimal effects. There were some images of blood work that showed this little phenomenon.
 
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I am going to check this out im curious ive heard this before but I didn’t see any proof so I believed that someone got drostanolone instead of EQ possibly and crashed there e2 because of to much drostanolone but now with this I want to look back at the action that they believe causes this to happen.
Im definitely going to take a look at Anabolics in a second and see if this is something that is explained.
 
👍. Interesting stuff. The downside is that finding quality literature is quite difficult most likely do the the demonization of AAS in general. If more widely accepted and utilized I would bet much more research would be out there, such that pubmed and other big journal article search engines would have more peer reviewed science to back up much of the information that can only be found through individual reports or websites. Basically the equivalent of using Wikipedia as opposed to pubmed to validate a claim. 😂 Fortunately, there are at least a decent body of literature out there, but not as much as I’d like to see.
 
Really no AAS research happens in the states in the UK and EU much more research is done and in Russia, China, and India they are definitely doing research we just don’t get to see it unless its a major publication in a serious medical journal which are controlled by you know who lol its almost like they don’t want the information to be available hmmm
 
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